Alexander Disease Research Update

Alexander Disease Research Update – Episode #32: 1) GFAP variants and oxidative stress, and 2) GFAP variants and lysosomes

Albee Messing

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0:00 | 32:18

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Albee Messing, Dolores Pérez-Sala, and Natasha Snider discuss the following recent publications: 

Alexander disease mutations differentially sensitize glial fibrillary acidic protein (GFAP) to posttranslational modifications and network disruption by oxidants.

                  Redox Biology 92:104103 (2026)

  Lysosomal function, resistance to oxidative stress and repair are compromised by expression of the Alexander disease GFAP R239C mutant.

                  Free Radical Biology & Medicine (in press)

Send your questions and feedback to:  amessing@wisc.edu

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